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Mechanisms that involve GSH as a cofactor in enzymatic reactions may be more crucial to the regulatory role GSH has on the redox environment than the capacity of the GSH/GSSG couple to act as a redox buffer [20]

The endogenous-acetylcholine-induced VEGF expression in neurons and astrocytes [302], the VEGF signaling system is in the rescuing effect of acetylcholine on NMDA-induced long-lasting hippocampal cell damage [303]

There is no known interaction with minoxidil at the topical level

Steady state cellular concentrations (M) and reaction velocities (M/hr) as S varies

10.1016/j.biocel.2011.12.013 113 HornL

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