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Spontaneous regression of tumour and the role of microbial infectionpossibilities for cancer treatment
In 2022, May et al
Glycogen synthase kinase 3 (GSK3) phosphorylates -catenin without Wnt signaling, which causes it to break down through a proteasome-dependent mechanism
doi: 10.1016/0012-1606(84)90201-x
(2011, American Journal of Pathology) reported that IGF-1 had plaque-stabilizing effects in a mouse atherosclerosis model useful as a half-life reference (~20-30 hours) and pathway marker
