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Firstly, this study is the first drug-target MR study to comprehensively investigate the specific impact of GLP-1RAs on SA, and we explored the possible mechanism of GLP-1RA through mediation analysis

Development of activity-based anorexia requires PKC- neurons in two central extended amygdala nuclei

GLP-1 is rapidly degraded by an enzyme named dipeptidyl peptidase-IV (DPP-4), converting to bioinactive products GLP-1 (9-36) and GLP-1 (9-37) ( GLP-1 exerts its function by binding to GLP-1R and is involved in the development and progression of many diseases ( GLP-1R agonists (GLP-1RAs) are a group of GLP-1 analogues that are resistant to DPP4-mediated degradation, working through activating GLP-1R and its downstream signaling ( The expression of GLP-1R remains controversial due to the lack of specific antibody against GLP-1R, with most of the literatures using qPCR to detect mRNA levels ( 2 GLP-1 in regulating immune system 2.1 The role of GLP-1 in innate immune cells Recent studies have demonstrated that GLP-1 and its analogues exert regulatory functions in innate immune cells, especially macrophages

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Women aged 30-49 (i.e., those most likely to be in perimenopause) are twice as likely to have been on a GLP-1 than their male peers