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Excessive production of ROS leads to oxidative damage that overwhelms the cellular repair mechanisms, resulting in mitochondrial dysfunction and the activation of apoptotic pathways [83,151,176]

Moreover, more than being a dead-end pathway the glyoxalase system may be important in the regulation of cerebral functions

UA-111573670-1

These results suggest that GliSODin is able to counteract the ability of HDM to induce Th2 and Th17 differentiation in T cells affecting on DC activation

However, the specific mechanism of NF-B regulation of SIRT1 still demands further exploration

A.ClementiE.MelaniM.BoverisA.CadenasE.MoncadaS.et al (2001)